With all of the lies and deception out there isn't it time you learned the truth, once and for all, about where you come from? I mean really?
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"If you get to the stage where you can persuade people on the evidence, that it's solid, that we are all African, that color is superficial, that stages of development of culture are all interactive," Leakey says, "then I think we have a chance of a world that will respond better to global challenges".Ok - we're good with that, and it's all quite sufficiently explained using the biblical model of a common created being fashioned by God only a few thousand years ago. Evolution theory gets no victory here. Leakey goes on in the article to blame humanity for what drives evolution - climate change - man made climate change no less.
"If you look back, the thing that strikes you, if you've got any sensitivity, is that extinction is the most common phenomena," Leakey says. "Extinction is always driven by environmental change. Environmental change is always driven by climate change. Man accelerated, if not created, planet change phenomena; I think we have to recognize that the future is by no means a very rosy one."Wait - what? Evolution theory dictates (and depends entirely on) that all currently living organisms are the off spring of extinct predecessors. Even Darwin wrote this in "On The Origins of Species". Leakey would have us to believe that extinction is "always" driven by environmental change and that man created it?
"Leakey began his work searching for fossils in the mid-1960s. His team unearthed a nearly complete 1.6-million-year-old skeleton in 1984 that became known as "Turkana Boy," the first known early human with long legs, short arms and a tall stature". - articleCreationists have nothing to fear from the Turkana Boy. Turkana Boy is our distant relative. But this is not because we evolved from the apes, but because Adam is the common ancestor of both Turkana Boy and ourselves. We should welcome the opportunity to discuss the scientific evidence through a biblical lens. Although museums and the popular media trumpet this human Turkana boy skeleton as an icon of evolution, a careful investigation reveals that the scientific evidence fully supports the biblical account. See here.
"I see no reason why you shouldn't go through your life thinking if you're a good citizen, you'll get a better future in the afterlife ...."Wait - I wonder if Leakey has ever actually read the Bible. Being a a good citizen to get a better future in the afterlife is the exact opposite of the general message given in the Bible which teaches that humanity is entirely incapable of being a good enough citizen to gain any favor at all in the afterlife.This is important because in the articles online about this Leakey mentions the book of Genesis directly.
"If you don't like the word evolution, I don't care what you call it, but life has changed. You can lay out all the fossils that have been collected and establish lineages that even a fool could work up. So the question is why, how does this happen? It's not covered by Genesis. There's no explanation for this change going back 500 million years in any book I've read from the lips of any God."

On the Origin of Species by Means of Natural Selection, or the Preservation of Favoured Races in the Struggle for LifeThe title changed to "On the Origin of Species" in the sixth edition. Darwin's book title unfortunately lends itself handily to the practice of racism among human-kind and has done so with many people like Hitler who famously taught that Jews were inferior enough to not even be considered human. Genocidal maniacs have oft quoted the standard evolutionary bias toward superior races or, as Darwin said, the "favoured" ones.
Like elephants and a few other animals, rhinoceroses are among the last of the large animals called "megafauna." Rhinos exist in African, Indonesian, Javan, Indian, and Sumatran varieties.1 They are also among the rarest animals in the wild.
The International Union for Conservation of Nature declared the Western Black Rhino officially extinct this year. The Northern White Rhino, also from Africa, may be on its way out as well, and the last Javan Rhino in Vietnam may be gone for good.2
Poachers are notorious for killing the large animals in order to harvest their highly valued horns, which some believe have medicinal properties, including use as an aphrodisiac. The horn is made of the protein keratin, the primary component of fingernails and hair.
This confirms what researchers generally observe about the extinction of species: The primary cause is interaction with humans. When mankind moves into an area, some of the first animals to face destruction are those that are the most threatening, such as the megafauna. Thus, recent centuries have seen the demise of such giants as Haast's eagle3 and the moa, a giant flightless bird.4
"The current extinction crisis is caused primarily by human impacts upon wild populations," according to UK biologist Rosie Woodroffe, who wrote on the declining populations of large carnivores.5
Rhinos are not carnivores, but in addition to being valued for their horns, they are large and can be dangerous if they are frightened or challenged. Thus, it stands to reason that if humans primarily cause the current extinctions of megafauna, then they may have caused past extinctions as well.6 That could help explain the plethora of legends, found in all cultures, of heroes killing dragons, which were probably dinosaurs in many cases.7
Why did the dinosaurs go extinct? One very likely factor—among others, such as climate changes after the Flood—is the same reason the black rhinoceros and so many other megafauna have died out: People moved in and eliminated them.
Bacterial cells are singularly long-lived. They keep dividing for what seems like forever. But because they are made of biochemicals, their DNA and proteins should suffer damage similar to what any other cell endures, including animal cells. What keeps bacterial cell components from wearing down?
Microbiologists have been trying to find out how these single-cell organisms handle chemical damage, which relentlessly accumulates due to friction and uncontrolled chemical reactions. So far, the results have been confusing, but a new analysis appears to have confirmed that bacteria have a remarkably well-engineered damage-reduction program.
University of California, San Diego biologist Lin Chao led a computer analysis of prior experiments.1 His team's work, published in Current Biology, cited a 2005 study showing that bacteria do age and that the cells do accumulate damage. But a subsequent study clearly showed no evidence of aging in the same bacteria species. Chao's analysis asserts that both are true.
His team proposed that when one bacterium divides into two cells, more of the damaged biochemicals end up in one than the other of the daughter cells. After many generations, a single population of bacteria ends up as a mixture in which cells filled with accumulated damage live side-by-side with "rejuvenated" cells.2
Chao said in a university press release:
So for a single celled organism that has acquired damage that cannot be repaired, which of the two alternatives is better—to split the cellular damage in equal amounts between the two daughters or to give one daughter all of the damage and the other none?2
"We think evolution drove this asymmetry," he said. But he did not explain how. He also said, "Because you have this asymmetry, one daughter by having more damage has aged, while the other daughter gets a rejuvenated start with less damage."2
Of course, the species as a whole will survive longer if each generation could redistribute damaged parts. But allocating so many tiny parts is a horrendous logistical problem.
Since no problem ever solves itself, either an intelligent person continually selects and removes the damaged biochemicals, or an intelligent engineer encoded an internal apparatus that identifies and transports the tiny offending chemicals into one daughter cell and not the other. There is no evidence that engineers live inside bacteria, so the latter option fits best.
While it makes sense that a dividing bacterium would give more damaged biochemicals to one cell than another, it makes no sense that "evolution"—which by definition excludes intelligent causes—could "drive" such a strategy. Strategies always come from strategists and never from nature.
Such an ingenious design could only have come from an ingenious Designer.
Evolutionary biologists argue that since human and chimp DNA are nearly identical, both species must have evolved from a common ancestor. However, creation scientists have pointed out that their DNA is, in fact, very dissimilar. The vast majority of each species' DNA sequence is not genes, but instead regulated gene expression. A new report unmistakably confirmed that the regulatory DNA of humans is totally different from that of chimps, revealing no hint of common ancestry.
Biologist John F. McDonald, of the Georgia Institute of Technology's School of Biology, and his team wrote that chimp and human genes are more than "98.5% identical," a commonly quoted statistic.1 Yet humans don't look or act 98.5 percent identical to chimps. Thus, something other than genes must be involved, and this has been overlooked in evolutionists' efforts to establish chimp-human ancestry. In 2005, molecular biologist and creation scientist Dan Criswell wrote:
However, such sequence similarity was based only on a fraction [less than four percent] of the total genome of man and chimpanzees, and reflects only the physiological similarities of humans and chimpanzees based on their cellular protein content, not the overall genomic content. The homology [similarity] frequently reported for the human/chimpanzee genomes excluded "indels," which are areas with zero sequence homology.2
"Indels" refer to insertions (in-) and deletions (-del) of genetic material, but they are simply DNA sequence differences.
Publishing in the open access journal Mobile DNA, the research team led by McDonald tested the hypothesis that the "substantial INDEL variation that exists between humans and chimpanzees may contribute significantly to the regulatory differences between the species."1McDonald said in a Georgia Tech press release:
Our findings are generally consistent with the notion that the morphological and behavioral differences between humans and chimpanzees are predominately due to differences in the regulation of genes rather than to differences in the sequence of the genes themselves.3
The team's analysis of indels confirmed exactly what Bible-believing biologists have been saying for years. The indels and other variously named non-gene DNA are not "junk DNA," and they are critical to the formation of each living creature.4 Biblical geneticist Jeff Tomkins wrote in 2009:
Most of the DNA sequence across the chromosomal region encompassing a gene is not used for protein coding, but rather for gene regulation, like the instructions in a recipe that specify what to do with the raw ingredients. The genetic information that is functional and regulatory is stored in "non-coding regions [including indels]," which areessential for the proper functioning of all cells, ensuring that the right genes are turned on or off at the right time in concert with other genes.5
The argument that chimp-human DNA similarity is evidence of common ancestry is possible only by ignoring the 98 percent of DNA that is different!6 It is like arguing that an aspirin pill is identical to a cyanide pill because they are the same shape and color. When do thedifferences enter the conversation?
Regulatory DNA—not just genes—is essential for each kind of organism, is almost entirely useful, and is different in humans than it is in chimps. How could billions of DNA differences have evolved in just four million years? It's impossible.7 Humans and chimpanzees were distinctly and uniquely created after all.
"The U.S. government has no evidence that any life exists outside our
planet, or that an extraterrestrial presence has contacted or engaged any member
of the human race," Phil Larson from the White House Office of Science and Technology Policy reported on the WhiteHouse.gov
website."In addition, there is no credible information to suggest that any
evidence is being hidden from the public’s eye."
Blue stragglers, according to NASA, "are older stars that acquire a new lease on life when they collide and merge with other stars."1 But a new study calls into question whether stellar collisions can account for these remarkable stars. And blue stars burn their fuel so quickly that they actually look young.
Since their discovery, evolutionary astronomers have sought a way to explain how these stars can even exist. They burn fuel so fast that they should have burned out billions of years ago. American astronomers Aaron Geller and Robert Mathieu published in Nature a description of their model for how older stars could have acquired a "new lease on life" by siphoning matter from nearby gas giant stars through "mass transfer."2
They investigated stars within a cluster called NGC 188, found in the constellation Cepheus. It contains 21 blue stragglers, 16 of which are binary stars that closely interact with nearby stars. The researchers suspected that the blue stragglers' partners were white dwarfs, which would be small, leftover remnants of larger red stars that the blue ones had drained of fuel. Such dwarfs are too faint for direct observation, but they have sufficient mass to cause their partner stars to wobble.
Of the 16 binaries, 12 had rotational periods right at 1,000 days and were thus called "long-period" blue stragglers. The study authors ran a statistical analysis that showed "the theoretical and observed [mass] distributions are indistinguishable."2 In other words, their theory that other stars "fed" these 12 blue stragglers matched well with what they observed.
But did this reconcile the relative youthfulness of these binary blue stars with their assumed billions of years of history? The answer is no. The authors wrote, "Blue straggler stars…should already have evolved into giant stars and stellar remnants,"2 and their new observations do not solve this deep-time problem.
Blue stragglers should burn through all their fuel in "a few million years at best."3 But these NGC 188 stars are supposed to be seven billion years old. So, to make them fit that age, these authors maintain that they were not initially blue stars, but instead burned fuel at a normal rate for billions of years. Then suddenly, within the last one million years, all 12 of them began siphoning extra fuel from their binary partners so that they only look young right now.
However, nothing explains the many blue stragglers that are not binary stars and yet exist near and far throughout the universe. Could they have received recent "youthfulness" through collisions with other stars?
In an article summarizing the Geller and Mathieu paper, University of Cambridge astronomer Christopher Tout wrote, "Thus, in this cluster [NGC 188], a collisional origin for blue stragglers is much rarer than expected, and the authors' study casts doubt on whether it occurs at all."4 The modeled scenario in which blue stars form by collision did not match observations.
So, isolated blue stars could not have received their young looks from a binary system, since by definition they have no binary from which to siphon fuel. They probably didn't receive their youthful appearance from collisions, either, according to these results. And though the binary blue stragglers may have siphoned fuel from nearby partners, the idea that 12 of 16 only did so recently—after an imagined 7-billion-year wait—defies reasonable odds.
Thus, the best explanation is still the most straightforward one—blue straggler stars look young because they are young.